Reference no: EM133858020

Injury control and trauma care technical expertise within government agencies, national and international professional societies, the World Health Organization (WHO), health institutes and academia could be leveraged to maximize the effect of health assistance for trauma care. Such action would result in the reduction of the injury burden and the strategic development of comprehensive emergency and trauma care systems.

1. How long should the statins be continued once the lipid profile returns to normal? Can we stop the statins once normal levels are attained and then continue with diet modification?

2. What is the exact mechanism of corneal arcus? What is its clinical significance? What is its relationship to hyperlipidaemia? Is there any effective treatment in medicine or alternative medicine to remove corneal arcus?

3. What is the best drug to be added to a statin in a case of familial hypercholesterolaemia not responding to lifestyle and diet modification plus a statin?

4. Can diabetes mellitus cause Horner's syndrome? If so, how?

5. Is it typical for a diabetic patient to experience angina during myocardial ischaemia?

6.  What are the causes of a flat oral glucose tolerance test (GTT)?

7. Are there any indications for routinely prescribing a statin (simvastatin, for example) in hypertensive and/or diabetic patients as prophylactic therapy?

8. What are the causes of vomiting and other gastrointestinal tract problems in type 2 diabetes mellitus? What is the correct treatment for a patient with nausea and vomiting, already taking hypoglycaemic agents and antihypertensives?

9. In diabetic ketoacidosis there is an overall potassium deficit. Since insulin promotes cellular potassium uptake, potassium should be given along with insulin in the treatment of ketoacidosis since hypokalaemia could potentially cause tachycardia. Why? I realise that hyperkalaemia can cause problems since the concentration gradient and therefore the membrane potential is reduced thus inhibiting proper function, but what is the problem with hypokalaemia? Is it that the membrane potential and the concentration gradient are too large? If so, how is this possible considering that the body may have too little potassium to make the intracellular concentration approach normal, so that although the extracellular concentration is reduced, the intracellular concentration is also reduced?

10. Can diabetes mellitus result in Horner's syndrome with no other neurological deficit?

11. Which drug is best recommended for a diabetic patient with rheumatoid arthritis resistant to methotrexate, and requiring frequent pushes with intramuscular depot preparations of methylprednisolone?