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Q. Can Pathophysiology causes cardiovascular disease?
Cigarette use activates platelets, increases circulating fibrinogen, increases heart rate, and elevates blood pressure. It appears to promote plaque disruption. A strong dose response relationship exists between smoke and CAD. Duration of smoking and the daily amount markedly influence risk of CAD. The adverse effect of smoking is present among men and women (but may be stronger in women) of all ages and ethnic groups with or without CAD. Data suggest that risk for cardiac death is two to four times greater among current smokers than non smokers.
Risk for cardiovascular disease begins to decline soon after smoking cessation, irrespective of age and sex. There is 50 per cent reduction in cardiovascular events within the first 2 to 4 years. It is thought to take as long as 20 years to regain baseline risk.
DNA binding domains called as basic domains rich in basic amino acids that was happen in transcription factors in combination with zipper helix-loop-helix or leucine (HLH) dimeriza
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what is the tolerance range of man
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What must be the basal clade?
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