Why does a mitral stenosis produce a loud s1

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Methods. Serial interviews with qualified nurses who were undertaking an educational programme or module that explored and examined models of nursing.

Findings. The outcome reveals that the use of the terms, models of nursing or nursing model, are limited and confusing as the terms can encompass a range of meanings. It is suggested that a three-model typology exists that clarifies more fully the present position of nursing models. The three models are Theoretical Model, Mental Model and Surrogate Model.

Limitations. The focus on data collection through serial interviews with qualified practitioners could have been broadened to include a range of data sources such as teachers and clinical areas, which could have enriched the phenomenon of models of nursing.

Question 1. Can carvedilol be used in an elderly patient who developed left ventricular failure after a very recent myocardial infarction, or should it wait until the condition becomes stable?

Question 2. I have seen some patients with chronic rheumatic heart disease with no obvious evidence of rheumatic fever (i.e. they could not recall if there was an episode of severe infection with subsequent fever and joint pain). So, actually, how is chronic rheumatic heart disease diagnosed?

Question 3. If a young patient presents with hemiparesis and rheumatic atrial fibrillation and is already on oral anticoagulant, with an international normalized ratio (INR) 3 and a normal computed tomography (CT)scan done 2 hours after onset, should he receive heparin for prophylaxis against further embolism? Should aspirin be combined with oral anticoagulant later, or should target INR be increased?

Question 4. When treating mitral stenosis using a balloon valvotomy, how come no thrombus develops at the site of the atrial septum or at the separated commissure of the valve leaflets?

Question 5. Why does a mitral stenosis produce a loud S1?

Question 6. Why is the mitral valve more affected than any other valve in the heart in most valvular diseases?

Question 7. What is William's syndrome (supravalvular obstruction)? Why does hypercalcaemia occur with this syndrome?

Question 8. Kindly tell me all the causes postulated for the collapsing pulse seen in aortic regurgitation.

Question 9. Does pulmonary stenosis cause pulmonary hypertension?

Question 10. Under what conditions would a pulsatile liver be found and what is its clinical significance?

Reference no: EM133858647

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