Increasing sympathetic input to the heart

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Reference no: EM133935223

Question

If someone states that "Increasing sympathetic input to the heart is not a mechanism for increasing CO to overcome increased afterload in the hypertensive patient." Evaluate this statement and make sure to explain why you do or do not agree with it. Feel free to read ahead and research "baroreceptor reflex" as you craft your evaluation.

You know that the work done by the heart (here, left ventricle) during ejection is (per stroke) ΔP X ΔV, where ΔP is change in ventricular pressure and ΔV is change in volume. How does this relationship, combined with LaPlace's Law, explain reduced stroke volume in dilated cardiac myopathy?

Reference no: EM133935223

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