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Describe roles of staphylococcal phenol-soluble modulin

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1. Describe the roles of staphylococcal phenol-soluble modulin (PSM) peptides, coagulase, membrane pore-forming toxins (such as leukotoxins, and leucocidins), superantigens, and staphylokinase play at different stages of skin wound infection and abscess formation and resolution by Staphylococcus aureus. Which of these virulence factors also play roles in the virulence of Staphylococcus epidermidis?

2. Describe a broad mechanism important to pathogenesis that is found uniquely in Gram(+) opportunists, but not in Gram(-) opportunists. Conversely, describe a broad mechanism important to pathogenesis that is found uniquely in Gram(-) opportunists, but not in Gram(+) opportunists. Please limit your answers to broad mechanisms, and not specific toxins or virulence factors. Describe how the mechanisms function in bacterial virulence and emphasize why they are unique to Gram(+) or Gram(-) bacteria.

3. Compare and contrast the virulence caused by Enterohemorrhagic E. coli (EHEC), and Uropathogenic E. coli (UPEC). Be sure to indicate host tissues affected, mechanism of interactions with host cells that make each specific to a different niche in the body, disease symptoms of infections, toxins produced, relative incidence of each disease in the USA, and outcomes of infections.

Reference no: EM133643988

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