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Q. What is Tyrosinemia?
There are two forms of hereditary tyrosinemia. They are tyrosinemia Type I and tyrosinemia Type II. Type I was thought to be due to a deficiency of parahydroxy phenylpyruvic acid oxidase. But recently secondary impairment in this enzyme has been attributed to a primary defect of hepatic firnaryl acetoacetate hydrolase. Patients with deficiency show renal tubular impairment, hypophosphatemic rickets, liver failure and hypertension. The plasma concentrations of phenylalanine and tyrosine are elevated.
In tyrosinemia type 11, there is a very elevated concentration of blood and urine tyrosine. Increase in urinary phenolic acids, N-acetyltyrosine and tyramine is seen.
Other symptoms include corneal erosions, hyperkeratosis on the fingers and palms and sole of the feet. Mental retardation may occur.
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Q. Symptoms of Angina Pectoris? The pain of angina is usually over the center of the chest (below the sternum) but can be felt from epigastrum to the jaw and arms. It is brough
Scope for public nutrition
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What is reduction? The most commonly understood definition of reduction is the removal of oxygen or addition of hydrogen ions. However, chemically, reduction can be defined as
Define the Role of Public Nutritionist in Health Care Delivery? It is clearly evident from the foregoing discussion that nutrition is an important, though not the only, determi
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Maturation Phase The Maturation phase characterizes he transformation of diploid primary spermatocytes into haploid spermatids. The primary spermatocyte divides meiotically t
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