Explain about the Glycemia or Insulinemia?

Preliminary research evidence indicates that there may be beneficial effects in terms of decreased fasting blood glucose/hepatic glucose production. This may be due to delayed gastric emptying, increased transit time, which may be dose-dependent. Alternatively, hepatic metabolism of glucose may be modified, mediated by the SCFA, especially propionate. Propioilate inhibits gluconeogenesis probably via its conversion to methylmalonyl CoA and succinyl CoA, both of which inhibit pyruvate carboxylase. Propionate enhances glycolysis and may lower plasma fatty acid concentrations.