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As humoral responses progress, the average affinity of the antibodies produced rises, in a process known as "affinity maturation." This seems at odds with at least one aspect of the clonal selection hypothesis, since the notion of "clonal integrity" implies that the progeny of an activated lymphocyte retain the specificity the original, pre-immune, antigen receptor. So here are the questions:
What is the basis for "affinity maturation" in humoral responses?
Does this contradict the tenets of clonal selection? Perhaps a bit more broadly, does this force rejection of the clonal selection hypothesis as a viable model to explain the cellular basis for specificity of adaptive immunity?
At the end of the response, you should raise an issue you are curious about related to the topic in the question.
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A state agency has contacted you to do a scientific assessment of kudzu in a nature preserve in southern Georgia. They are concerned about the effects of kudzu.
In horses, the black (B) allele for hair color is dominant over the brown (b) allele and the trotter (T) allele for the two beat gait is dominant over the pacer (t) allele. A black, pacer horse mates with a brown, trotter horse producing a brown, pac..
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What experiment could be performed to ascertain if an antimicrobial agent is bactericidal or bacteriostatic? Can a bacteriostatic agent become bactericidal in the long run or vice versa? Why?
What is the advantage of using ATP as a common energy source? Another way of asking this question is, Why is ATP an advantage over using a bunch of different food molecules?
A monoprotic acid, HA dissolved in water and the equilibrium concentrations of the reactants and products calculate the value of pKa for the acid HA
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