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Protecting the tomcod

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If the PCB toxicity mutation is so effective at protecting the tomcod, why don't all the tomcod in all populations have it? I already explained how selection pressure plays a role but I need help explaining the second aspect. I don't want an answer word for word I just want to be steered in the right direction so I can look in my notes and apply the right information.

Reference no: EM132510641

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