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Huntington disease is a fatal nervous system disorder causing degeneration of the brain. It is caused by a defective gene on chromosome #4. This is an autosomal dominant trait affecting 1 in 20,000 people. However, it is a delayed action gene that is not expressed until the affected individualis in his or her late 30s or40s. The homosygous condition is lethal to the fetus. Using the Hardy-Weinberg equation, determine (1) the frequency of carriers in the population, (2) the frequency of fetuses affected by the lethal homozygosity, and (3) the frequency of unaffected individuals.
define how the axonal membrane potential is restored. Describe how resting ionic distributions are maintained.
analysis of restriction fragment length polymorphisms can be used to distinguish between alleles based on differences in which of the following.
Determine which of the following organelles is directly connected to the outer membrane of the nucleus in a eukaryotic cell?
q1. why do mosses and ferns both require free water to complete their life cycles? at what stage of the life cycle is
What is meant by the term "vicariance"?
Compute the ratio of molecular weights of 60S and 40S ribosomal sub-units assuming that sub-units have spherical shape and the same density.
How is it that different cells can respond in different ways to exactly the same signaling molecule even when they have identical receptors?
q1. why is determining the epidemiology and ecology of disease important to learning about infectious diseases?q2.
Describe principle behind various whole genome sequencing technologies. Describe application of one whole genome sequencing from literature for providing deeper insight into biological function.
Polio and HIV are both +ssRNA viruses have very different strategies for replicating their genomes. After the viral genomes enter the cell, explain the first step involved in replication of each genome.
you have just been hired as an information security engineer for a large multi-international corporation. unfortunately
On the basis of these results, advance a hypothesis about the cause of the phenotype. Based on your hypothesis, what do you predict would happen to developing T cell in an HD mouse which has been made transgenic for a TCR restricted to I- A b .
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