How are des-loratidine and levo-cetirizine different from

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Question 1: While auscultating, I have been instructed to examine vocal resonance by asking the patient to utter the number ninety-nine. Over an area of consolidation, I listen for a louder "ninety-nine" and quieter sounds with effusion. Is this accurate?

Question 2: Which job does bupropion play in quitting any pretense of smoking?

Question 3: The clinical signs and symptoms of rhinitis and the common cold (influenza) are very similar. How can I tell them apart from one another?

Question 4: How are des-loratidine and levo-cetirizine different from their parent compounds? Are they safe for breastfeeding and pregnancy?

Question 5: What distinguishes pneumonia from acute bronchitis? Are both illnesses brought on by an infection? 156 14 Diseases of the respiratory system Diseases of the respiratory system 14 157 Should oral steroids, such as prednisolone, be discontinued until the patient's liver function improves in the event that a chronic bronchitis patient develops obstructive jaundice and Escherichia coli biliary sepsis? 2. During the patient's treatment with IV ciprofloxacin, gentamicin, metronidazole, and cefuroxime, are there any adverse reactions that prevent the patient from taking steroids concurrently?

Question 6: of Robbins Basic Pathology, patients with chronic obstructive pulmonary disease (COPD) have either a normal or slightly elevated forced ventilation capacity (FVC). That just doesn't make sense to me. I mean that it ought to go down. And your book specifically mentions this. Since I was unable to get in touch with the authors of that book, I decided to inquire about the possibility of an increase in the FVC in COPD patients.

Question 7: Is there a specific obstructive pulmonary condition that might result in a higher FVC? By what means, if so?

Question 8: I'm not sure if clubbing is a sign of chronic obstructive pulmonary disease (COPD). You've said that it isn't, but some books say that clubbing is a sign of COPD.

Question 9: A 70-year-old man with chronic obstructive pulmonary disease (COPD), a history of myocardial infarction, and a 25% left ventricular ejection fraction (LVEF) is dyspneic when doing something as simple as walking or taking a bath. He claims to have no paroxysmal nocturnal dyspnoea (PND) and is not orthopneic. He has normal blood pressure and does not wheeze or cough. Amiodarone has been prescribed to him for the past year, and he has had three episodes of ventricular tachycardia (VT). What is the most effective method for determining the precise cause of this dyspnea? If the airways are already dilated, why should a bronchodilator be used in bronchiectasis?

Reference no: EM133446025

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