Pathophysiology 

The exact mechanism of glomerulonephritis  is still under investigation. The dies'ease is characterised by deposition  of immune complexes  consisting of antigen antibody reaction and some components  of complements. These mateials become  trapped in glomerular capillary loops. The glomerulus  become edematous  and bloodless with the resulting infiltration process  and there is decrease  in filtration of plasma. This decrease  in filtration results in increased retention of water and sodium which further  leads to circulatory congestion and edema.